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TMP269, a potent and selective class IIa HDACs inhibitor, significantly improves the pathological changes including renal tubular dilation, cell debris accumulation in the renal tubular lumen, interstitial edema, and inflammatory cell infiltration in FA-/IRI-induced AKI mouse model by reducing the expression of HDAC4 to regulate pathways associated with apoptosis, autophagy, and proliferation

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This study design gives researchers confidence that any improvement is due to the treatment, not other factors

SIN has therapeutic effects in streptozotocin-induced gestational diabetes mellitus (GDM) rats with high safety, which is associated with the inhibition of inflammation and oxidative stress via the TLR4/MyD88/NF-B signaling pathway (130)