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The dominant driver is reduced caloric intake

The aim of this review is to summarize the increasing list of roles of IGF-I, both in physiological and pathological conditions, underlying that its potential therapeutical options seem to be limited to those proven states of local or systemic IGF-I deficiency as a replacement treatment, rather than increasing its level upper the normal range

Instead, lipidated LC3 accumulated in ATXN3 KO cells at later stages compared to parental cells, and this was not further increased by chloroquine treatment, showing that lysophagic flux was compromised in ATXN3 KO cells (Fig