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The best example is the mechanisms for downregulating HMGB1, which we could deduce to be a therapeutic target for alleviating inflammatory reactions and thereby the associated DCM, particularly the myocardial damage evidenced by the rodent models utilized in the present study

The amounts of GSH and protein thiol increase significantly and the mRNA levels of gamma-glutamylcysteine synthetase and metallothionein are significantly higher in UDCA-treated hepatocytes than in controls [77]

Role of catalase in oxidative stress -and age-associated degenerative diseases