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Description

In the absence of any clinical evidence that the apparent congenital cyanosis was due to cardiac or lung disease, inherited methemoglobinemia was considered and confirmed with the finding of a methemoglobin of 40 % and absence of the red-cell enzyme cytochrome b5 reductase (Type I deficiency)

Again, only the 5-containing C19 peptide showed strong binding with Ufd1

Nephrology 23, 867875

After downregulation of Nrf2 or HO-1, the protective effect of JSH-23 against the H 2 O 2 -induced reductions in the levels of osteoblast markers (Runx2, OCN and BMP2) was attenuated (Figures 7K,L)