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Stress response and genome maintenance pathways and cancer FDA-approved anticancer therapies targeting Bcl-2, PARP, and CDK4/6 While targeting stress response and genome maintenance pathways (e.g., ATM, ATR, p53, Bcl-2 and PARP) for anti-aging interventions remains largely confined to preclinical studies in cellular and animal models, the therapeutic exploitation of these same pathways in oncology has achieved significant clinical translation

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4-9 before with the effect of description afterwards.Main curative effect terminal point is the percentage ratio the experimenter of the complete reaction that has the negative Histological assessment that is defined as the target damage the 6th week.The second curative effect terminal point (Secondary efficacy endpoint) is reflected in the percentage ratio the experimenter of at least 50% minimizing that has the area (products of two full diameters) that is defined as the damage of being treated the 6th week.Said result shows that 23.5% ITT colony has complete reaction in the 6th week, and 18.5% PP colony has complete reaction in the 6th week