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In -synuclein-induced PD mouse model, Zhang and collaborators showed that the administration of caffeic acid prevents the neurodegeneration of dopaminergic neurons in SN and improves behavioral abnormalities by stimulating autophagy through the JNK/BcL2 pathway (Zhang et al., 2019)

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HepG2 cells develop signs of riboflavin deficiency within 4 days of culture in riboflavin-deficient medium

Since callus proliferation was higher in leaf explants, subsequent experiments were carried out on leaf explants