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Next, we assessed the pulse-chase labeling of ITalk

179 The results from high-resolution phosphor imaging autoradiography established the specific binding of IGF1 and binding sites

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Silencing of Nrf2 by siRNA (siNrf2) significantly attenuated the allicin-induced increase in cell viability and HO-1 and decrease in NLRP3 protein in LPS-stimulated H9C2 cells [25]