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found that selenium deficiency in RAW 264.7 macrophages increases oxidative stress and enhances iNOS expression and NO production through heightened NF-B activation, suggesting seleniums key role in regulating inflammatory responses (Prabhu et al., 2002)

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The activated iodine binds to tyrosine residues on thyroglobulin molecules under the action of TPO to produce monoiodotyrosine (MIT) and diiodotyrosine (DIT), which are subsequently coupled to produce T3 or T4 ( Figure 1 )