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Nevertheless, parkins contribution to H 2 O 2 reduction in the cytosol is unmasked under the following conditions: (i) in the presence of redox stressors, either due to a second genetic hit in mice (Figs

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A stack combining acute neuroprotection (BPC-157), broad neurotrophic support (Cerebrolysin), and long-term neurogenesis (P21) could theoretically address the full timeline of TBI recovery

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