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Ample of proteomics and metabolomics studies have proved the considerable lipid metabolism remodeling in the myocardium of AF patients ( 3 AR) ( 2+ -handling and cell contraction, mitochondrial biogenesis, cell growth and proliferation, and so on, thus exerting cardiovascular protection with a combination of multiple mechanisms ( via LCA, a natural and biologically active micronutrient enhancing physiological FAO through CPT1B (Figures 3, 6), can significantly reduce obesity-mediated AF propensity and the corresponding atrial remodeling (Figures 1, 2), thus better supports the efficiency of FAO promotion in the AF therapeutic approach

Such recombination between the two mutated specific nucleic acid sequences will result in the production of double or triple hybrids in some situations