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4 Conclusion and perspectives Although human kidney diseases arise from complicated pathological mechanisms and different etiologies, mitochondrial dysfunction is no doubt plays an important role in the pathogenesis

While supportive evidence has been obtained from animal models and preliminary clinical studies, further large-scale, long-term randomized controlled trials are imperative to establish its safety and evaluate its clinical efficacy comprehensively

Exploring the imbalance of circulating follicular helper CD4+ T cells in sarcoidosis patients

Tyrosinemia associated with hypermethioninemia and islet cell hyperplasia