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5 Conclusion Owing to the complex aetiology and recurrent symptoms of UC, developing innovative treatment strategies that can repair mucosal barrier function, promote deep mucosal healing, and effectively maintain remission is a major challenge

doi: 10.1007/s00726-003-0011-2 55 AugustyniakEAdamAWojdylaKRogowska-WrzesinskaAWillettsRKorkmazAet al

For instance, one study has demonstrated that endogenous DNMT3A and DNMT3B are associated with HDAC activity, where DNMT3A facilitates transcriptional repression via specific molecular interactions between its ATRX-like PHD domain and both HDAC1 and the co-repressor RP58 (302)

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