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Results Contractile recovery following metabolic inhibition is reduced but cell survival is unaffected by TMAO Given the association of high TMAO concentrations in the blood and the incidence of cardiac disturbance in patients, it was hypothesised that increasing concentrations of TMAO would worsen the outcome for cardiac cells in a widely used metabolic inhibition and simulated reperfusion (MI/R) model (Figure 1A) ( ATP channel, Kir6.2/SUR2A, as an indicator of severe ATP depletion (Figure 1B)

mit dem Urin ber die Nieren ausgeschieden , um eine stabile Konzentration im Blut aufrechtzuerhalten

The other problem is that if the waste is not cleared out, those toxic energy production by-products can cause vasoconstriction (narrowing of the arteries)

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