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Turtola M

365,366 Collectively, these studies provide strong support for the CYP4:20-HETE pathway as a potential drug target for combating tumor growth and metastasis

Neoplastic cells may evolve adaptive pathways to circumvent drug-induced cytotoxicity, including enhanced drug efflux, DNA repair activation, and apoptotic signaling dysregulation (Patel and Kumar, 2025