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Semax works primarily through BDNF receptor (TrkB) modulation and immediate neurotransmitter effects (particularly dopamine and serotonin)it has human clinical trial data from Russian studies and rapid-onset cognitive effects

Along with the upregulation of TLR2 and TLR4, HMGB1 release and NF-B activation were observed with increased cytokine expression in PTECs upon exposure to high glucose for 72h

These doses sit well within the range tested in animal safety studies and far below levels where any toxicity was observed

The practice of prescribing this compound shows an utter lack of respect for patient health and safety