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Cystine starvation has been shown to impede TNBC growth, which affects stem cell properties and chemotherapy resistance in TNBC (27), thereby promoting TNBC cell ferroptosis through activation of the general control nonderepessible 2-eukaryotic translation initiation factor 2 subunit 1-activating transcription factor 4-CHAC1 pathway through the specific cytosolic GSH degradation enzyme CHAC1 (28)

In normal physiological conditions, the organelle utilizes a variety of mechanisms to tightly regulate ROS (25)

Drug loading and encapsulation efficiency rose to 56% and 86%, respectively, owing to the presence of AgNPs

43: 26558 Tariq M, Morais C, Sobki S, et al