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Results HIF1 and HIF2 KO NSCLC cells are metabolically rewired towards ser/gly synthesis To investigate the metabolic differences between HIF1 and HIF2 under normoxic conditions, we used NCI-H1299 lung cancer cells with HIF loss-of-function mutations that were previously generated in our group, i.e., HIF1 KO and HIF2 KO, using HIF wild type (WT) as control [34]

This paper demonstrates that the genetic architecture of ALS is largely based on rare variants and that powerful new genetic insights can arise through the analysis of large numbers of cases available through the Project MinE consortium

This allows the application of pivotal quantities and the corresponding reference distributions to diagnose model inadequacy at any level of a hierarchical model

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