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Introduction Abnormal glutamate (Glu) brain levels and function of the glutamatergic system have been found in different psychiatric disorders 14 and neurological diseases including leukodystrophies and mitochondrial disorders 5 , epilepsy 6,7 and Huntingtons disease 8 to just name a few

Alternatively, the increased longevity could have resulted from decreased product-inhibition of the nematode homolog of cytoplasmic GPD1 by decreasing glycerol-3-phosphate levels, leading to increased flux through GPD1 to oxidize the cytoplasmic [NAD + ]/[NADH], a known prolongevity intervention (Lautrup et al., 2024)

Hampel H, Hardy J, Blennow K, Chen C, Perry G, Kim SH, Villemagne VL, Aisen P, Vendruscolo M, Iwatsubo T, Masters CL, Cho M, Lannfelt L, Cummings JL, Vergallo A, et al