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Primarily, PHOSPHO1 curtails the progression of ferroptosis by dampening PE levels within the endoplasmic reticulum (ER), effectively inhibiting lipid peroxidation

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Although ferroptosis shows great potential in B cell immunomodulation and tumor therapy, the current treatment of DLBCL still faces significant challenges: the sensitivity of the GCB subtype to ferroptosis and the resistance of the ABC subtype create a therapeutic dilemma

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