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Li, J., Qu, X., Tian, J., Zhang, J

It is therefore likely that GSTP-silencing may directly elicit mitochondrial dysfunction, resulting in the rapid generation of ROS, which is reasonable because the primary source of ROS is mitochondria

These changes disrupt network activity, with mPFC cells showing reduced phase locking and altered preferred phases, thereby slowing in vivo mPFC local field potential (LFP) oscillations, which may underlie autism-related phenotypes (104)