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Plasma lipids and lipoproteins in Japanese male patients with coronary artery disease and in their relatives

The relative contribution of this pathway to disulfide formation is not clear but it is known that combined knockdown of Ero1, PrxIV, and VKOR compromises disulfide formation and causes cell death in a mammalian cell culture system (Rutkevich and Williams 2012)

Dis Model Mech 3:652662 ahin A, Held A, Bredvik K et al (2017) Human SOD1 ALS mutations in a drosophila knock-in model cause severe phenotypes and reveal dosage-sensitive gain and loss of function components