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6 Conclusion In conclusion, lactylation modification represents a fundamental mechanistic bridge coupling tumor metabolic reprogramming with epigenetic regulation, providing a conceptual framework for understanding broad-spectrum therapy resistance in cancer

Production costs are also considered a major limit

This function is particularly relevant in neurons, where sHSPs frequently colocalize with disease-associated protein aggregates and serve as an early line of defense against proteotoxic stress

These findings appear to be consistent with FRs ability to maintain cells in an undifferentiated state during development