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Within this network, methylenetetrahydrofolate reductase (MTHFR) reduces 5,10-methylene-THF to 5-methyl-THF, enabling remethylation of homocysteine to methionine and sustaining S-adenosylmethionine (SAM), the universal methyl donor

ATOX1 upregulation enhances genotoxic drug resistance and promotes cancer cell migration by copper-dependent activation of mediator of DNA damage checkpoint 1 (MDC1) and lysyl oxidase (LOX) proenzyme [83, 84]

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Most Common Side Effects Nausea Constipation Diarrhea Decreased appetite Occasional vomiting These effects typically occur during the first few weeks of therapy and tend to resolve as the body adapts to the medication