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To bridge this gap, the present review aims to provide the current understanding of cuproptosis, with an emphasis on its anticancer activities through the interaction with copper-induced oxidative stress, thereby getting ideas for future research directions in this field

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Oxidative stress and neurodegeneration: where are we now

Copper directly binds with the lipoylated proteins of the TCA cycle, leading to the aggregation of these lipoylated proteins and disruption of the iron-sulfur cluster proteins and consequently proteotoxic stress [3]