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Mutations in SLC33A1 cause a lethal autosomal-recessive disorder with congenital cataracts, hearing loss, and low serum copper and ceruloplasmin

Importantly, overexpressing FTMT, a mitochondrial iron chelating protein, largely inhibited oxidative stress-induced ferroptosis, further linking mitochondria iron overload to ferroptosis

Each has documented research effects in mammalian models with relevance to aging physiology

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