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To date, metabolomic studies have identified quiescent-stage signatures of ulcerative colitis and have demonstrated the potential of metabolomics in predicting both disease relapse and biologic agent efficacy [580]

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Inhibition of MCAD in GBM cells (either genetically or pharmacologically) leads to the accumulation of medium-chain FAs, lipid peroxidation, and mitochondrial damage, resulting in apoptosis [124].An interesting use of FAO by glioma cells is to evade anti-tumor responses induced by radiotherapy

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