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However, unlike pro-fibrotic agents, GHK-Cu also upregulates the activity of matrix metalloproteinases (MMPs) and their inhibitors (TIMPs), facilitating the breakdown of scarred or disorganized collagen

It is noteworthy that during the early stage of certain extra-intestinal organ injuries, there is an increase in intestinal permeability and subsequent release of endotoxins, which plays a crucial role in the timely intervention of the disease

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Specific inhibition of HK2 with 3-bromopyruvate reduces joint swelling and histological damage in SKG mice, a RA model, by suppressing glycolysis-dependent dendritic cell activation and Th17/Treg imbalance