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Recent evidence underscores that CKD triggers persistent immune activation through uremic toxins, oxidative stress (OS), and microbial imbalances, fostering an inflammatory environment that exacerbates bone resorption and VC, hallmark features of CKD-MBD ( This review examines advances in understanding the immune mechanisms of CKD-MBD and explores emerging therapies to mitigate its clinical burden

56 Foxp3 expression and Treg stability are governed by multiple molecular mechanisms, including transcription, translation, epigenetic modifications, and posttranslational regulation

However, it is a challenge to determine -Syn in CSF, depending on careful sample collection and processing

Obesity and the metabolic syndrome in children and adolescents