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The GHK-Cu complex depends on the copper(II) oxidation state to maintain its coordination geometry and the biological activity attributed to that complex

However, multiple limitations in preclinical studies reduce the significance of current findings

These include modulation of pathways involved in inflammation, oxidative stress, epigenetics, mitochondrial dysfunction, the gut microbiota, tryptophankynurenine metabolism, the HPA axis, neurogenesis and BDNF, epigenetics, and obesity