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3 rd ed

Growth hormone stimulates hepatic IGF-1 production by binding to growth hormone receptors on liver cells, triggering intracellular signaling through the JAK2/STAT5 pathway that upregulates IGF-1 gene expression

Despite these divergent pathways, both sexes converged on the same endpoint: a desperate, compensatory upregulation of intracellular glutathione in immune cells that ultimately drains the hosts systemic energy