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The subsequent formation of PAOs (protein amyloidosis precursorsprotein oligomers) and polymerization deposition lead to the progression of amyloidosis in cardiomyocytes, increasing the risk of AF and heart failure [37, 66, 69]

Several studies chose to validate their defined signatures in a secondary dataset and noted improved prediction of outcomes when metabolic biomarkers were used with protein biomarkers (S100b, UCH-L1, GFAP, etc.) [30, 31]

In the context of inflammation and disease, an intriguing division of labor occurs between microglia and astrocytes with microglia (largely) increasing phagocytic capacity, while some sub-states of reactive astrocytes shutting down phagocytosis almost completely [264]