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Thus, the effect of CK1, PKC, and p38 MAPK varies from pro-survival in some erythrocyte precursors to pro-eryptotic in mature cells, which might be a consequence of missing downstream effectors in circulating erythrocytes

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These drugs target cells in the cochlea, mostly hair cells, by increasing oxidative stress and induction of cochlear inflammatory processes and cell death, and generally produce permanent hearing loss

In contrast, it showed a higher glutamate level in the brain in a mouse model of Parkinsons disease [141]