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USA 108 , 14151420 (2011)

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DOI:10.1016/j.aohep.2025.101777 21

For instance, PKC ) or overexpression of a cleavage-resistant PKC mutant protected against METH-elicited apoptosis in mesencephalic dopaminergic cell cultures in vitro ( PKC inhibition may rescue METH-elicited mitochondrial burden, pro-apoptosis, and dopaminergic degeneration, implying that PKC is an important gene involved in METH-induced mitochondrial dysfunction and apoptosis in dopaminergic neuronal cells ( Recently, Zhang et al