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Of relevance, recent evidence indicates that SAM exerts anti-inflammatory effects by reducing the expression of the proinflammatory cytokine tumor necrosis factor-alpha (TNF) and increasing the expression of the anti-inflammatory cytokine interleukin 10 (IL-10)

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data, after adjusting for underascertainment of infections and biased assumptions on post-hospitalization vaccination rates, exposed gross overestimates of the infection-related myocarditis risk for unvaccinated males under 40