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Eur Heart J 38:557564

For instance, p53 can demonstrate antioxidant activity at physiological ROS levels to preserve cellular integrity but paradoxically amplifies ROS under oxidative stress to eliminate irreparably damaged cells while protecting neighboring tissues [69]

who resided in or traveled in regions where mycoses are endemic, 5

Similarly, CRP may affect the production and function of MCP-1, highlighting the intricate network of inflammatory mediators involved in the pathogenesis of CHD (138)

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