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In contrast, higher concentrations inhibit mitochondrial complex I, thereby reducing oxidative stress and delaying the opening of the mitochondrial permeability transition pore (mPTP), a key event in ischemia-reperfusion injury (Zilov et al., 2019)

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Glucocorticoids also induce the expression of MAPK phosphatase-1 (MKP-1), which attenuates MAPK signaling cascades, further lowering inflammation and fibroblast activation [128]