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Drugs that target intrinsic metabolic vulnerabilities (e.g., GLUT1 inhibitors, PDK4 inhibitors, or glutaminase inhibitors) predispose cancer cells to death, which is triggered by decreased nicotinamide adenine dinucleotide phosphate generation or increased reactive oxygen species accumulation

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Still, confirming the clinical relevance of newly developed antioxidants will require a paradigm shift in the testing approaches

Remarkably, not only did resveratrol restore normal cell cycle function earlier, but DNA strand breakage repair was detectable sooner in irradiated cells pretreated with resveratrol [33]