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Moreover, these exosomes partially inhibit premature activation and depletion of primordial follicles by modulating the PI3K/AKT/mTOR signaling pathway, which is typically hyperactivated by chemotherapy, resulting in follicle burnout. Following treatment with hAEC exosomes, there is a reduction in the phosphorylation levels of critical proteins, including Akt and FoxO3a, which contributes to the preservation of ovarian reserve

Neuroinflammation disrupts neurotransmitter systems, reduces neurotrophic support (BDNF) and alters structure and function in mood-relevant brain areas (hippocampus, amygdala, prefrontal cortex, striatum)

Gut microbiome and polycystic ovary syndrome: interplay of associated microbial-metabolite pathways and therapeutic strategies

Prokop, M., Damborsky, J