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Modeling monogenic diabetes using human ESCs reveals developmental and metabolic deficiencies caused by mutations in HNF1A

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A solution containing 1.75 mg/ml fibrinogen and 50 nM FXIIIa in 50 mM HEPES buffer containing 5 mM CaCl 2 was incubated with different concentrations of EA (100, 500, 1000, 3000, and 5000 M)