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To compensate, glycolysis was accelerated, and spare glycolytic capacity was markedly reduced to maintain bioenergetic homeostasis [8]

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This functional response, which also enhances the phagocytosis of apoptotic cells and collagen deposition, serves to protect the host from inflammatory damage and promote tissue healing ( During the initial phase of mycobacterium tuberculosis infection, the expression of IFN- and iNOS in the BALF of mice gradually increased, aligning with the observed trend of AM polarization, indicating M1-biased AM polarization