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Additionally, busulfan can decrease Bcl-2 gene expression (as an antiapoptotic gene) and increase Bax gene expression (as a proapoptotic gene) in epididymal sperm of busulfan treated mice, which are related to increased oxidative stress as a result of decreased enzymatic antioxidants of GPx and SOD and increased level of lipid peroxidation in sperm cell (50)

Study drug supply was performed at baseline visit and after 3 months of treatment

No significant adverse effects were reported in both the placebo and intervention groups (Table 3)

Chronic exposure to an irritant (e.g., cigarette smoke) causes inflammatory cells (e.g., neutrophils, macrophages, eosinophils) to gather in the airspaces of the lung