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Conclusion Our work demonstrated that OS can cause endothelial-mesenchymal transition by inhibiting mitochondrial autophagy and promoting apoptosis in silicosis

Based on preclinical literature reviews, Pickart and colleagues proposed broader genomic effects, suggesting that, in sequencing models, GHK-Cu may influence approximately 31% of genes altered during skin aging, shifting expression patterns toward a younger phenotype [1]

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Similarly, studies in aged rats showed that sevoflurane and isoflurane contributed to PND by promoting the release of proinflammatory cytokines and activating microglia in the brain (Peng et al., 2012