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Over weeks and months, this pattern of stress response reactivation changes the equilibrium of essential hormones that regulate ovulation, sperm production, and sexual function

At the molecular level, these changes are principally determined by hypoxia-inducible factor (HIF)mediated reshaping of the transcriptional profile, which depends on the extent, duration, and severity of hypoxia and by unfolded protein response (UPR)modified translational program activated in conditions of severe hypoxia/anoxia [4, 5]

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Dysregulation of TrkB receptors and BDNF function by amyloid- peptide is mediated by calpain