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By interrupting their interaction, were effectively collapsing the structural foundation that allows fibrosis to progress. Unlike traditional therapies that attempt to inhibit FAKs enzymatic activity often with poor specificity and side effects FN-2012 works by disrupting protein-protein binding

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This suggests that M pro might be particularly efficient at disrupting GSH synthesis, as compared to action at some of the other target sites predicted here, thereby contributing to the clinical findings in COVID-19 ( Finally (Figure 2D), we include a non-canonical hit that would cleave at position 90 of glutaredoxin-1 (GLRX-1)

They might also feel drowsy, or some other metabolic function might change slightly