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In diabetic patients, metabolic abnormalities can lead to alterations in amino acid signaling pathways, which, in turn, affect mTORC1 activity and disrupt the autophagy process (Singh et al., 2024)

Coupled with endogenous reactive oxygen species (ROS) generated as by-products of various cellular processes, strict regulation of antioxidants is critical to maintaining a balanced reduction/oxidation (redox) state

The number of toxins our body has to deal with every day is truly remarkable

To evaluate whether mutants deficient in different glutathione-dependent enzymes are impacted by TMHT, we subjected cytosolic glutaredoxin mutants to TMHT (Figure 4C)