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Given that one of the severely damaged regions following TBI is the cerebellar circuit, PQQ could stimulates neurite regeneration in a wound recovery model using cultured cerebellar granular neurons (CGNs) (Shanan et al., 2019)

This response could be caused by an increase in ROS or ammonia, which aged explants cannot process as effectively due to lower GSH production

Rheumatoid arthritis reprograms circadian output pathways

Indeed, lack of consistent CD-like enteropathy in humanized mice [61] supports the concept that the accelerated disruption of enterocytes secondary to the adaptive CD4 + T cell insult cannot fully explain CD pathogenesis, supporting the notion that an intrinsic defect of the stem cell compartment in subjects at risk of CD is a key element of CD enteropathy [60, 62]