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Non-glutathione disulfide substrates that employ monothiol mechanism (iii) should not be trapped as Grx(SSR) intermediates, which might explain the rather small number of verified non-glutathione disulfide substrates of glutaredoxins to date

Controlling ferroptosis determines whether cells proliferate or die

This relationship can be rationalized by the similar chemical mechanism, involving an aromatic nucleophilic attack by the sulfur of glutathione to form a Meisenheimer -complex, which is followed by the release of the common conjugate S-2,4-dinitrophenylglutathione

There is evidence of increased gut permeability in AD, which might allow more pro-inflammatory molecules to circulate [127]