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Hippocampal subfield vulnerability to -synuclein pathology precedes neurodegeneration and cognitive dysfunction

There is a weak association between lipid metabolism and NETosis levels under physiological conditions, as NETosis usually occurs in mature neutrophils with little reliance on lipid metabolism for energy production (148, 149)

A key early component of this response is the autocrine and paracrine production of interleukin (IL)-2, which acts as a critical driver of T cell proliferation and effector differentiation by supporting expansion of antigen-specific clones and promoting the survival of effector T cells through STAT5 signaling 2,3,4,5