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Extended-interval dosing is associated with equivalent efficacy and reduced nephrotoxicity, with a non-significant trend toward lower ototoxicity in pooled analyses

Importantly, the dependency on riboflavin and subsequently impaired oxidative phosphorylation upon perturbation of riboflavin metabolism can be exploited by sensitizing AMLs to the BCL-2 inhibitor venetoclax

Beyond its classical metabolic role, research has found that DHODH can limit lipid peroxidation within mitochondria by maintaining its own mitochondrial CoQ/CoQH 2 redox pool, thereby independently suppressing ferroptosis (Mao et al., 2021

Patel VI, Booth JL, Dozmorov M, Brown BR, Metcalf JP