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Mutations in MMAA , MMAB , and MUT (corresponding to cblA, cblB, and mut complementation groups) are associated with isolated methylmalonic aciduria, while mutations in MTRR and MTR (corresponding to cblE and cblG complementation groups) are associated with isolated homocystinuria

VRK2 overexpression could suppress the increase of shrunken mitochondria and lipid ROS induced by TBHP, while BFA treatment abolished VRK2-mediated alterations (Fig

Metabolite information of the longissimus dorsi muscle in positive ion mode